Further animal studies have demonstrated that short-term osteocalcin treatment in mice can mitigate hepatic steatosis.[35] Furthermore, bone harbors a substantial population of mesenchymal stem cells, which have been shown to modulate CD4 T cell differentiation in murine models, consequently alleviating nonalcoholic liver steatosis.[36] Although there is currently no clear research on the relationship between BMD and liver enzymes, the above indirect findings may help partially explain the relationship between BMD and ALT. The gene discussed is CD4; the disease is Hepatic steatosis.