found that inhibiting the phosphorylation of the Src family could regulate neutrophil inflammation.[36] Meanwhile, in a rat model of osteoarthritis, Src activates the signal transducer and activators of transcription 3 (STAT3)/activated mitogen‐activated protein kinase (MAPK) pathway, exacerbating the tissue damage.[36] Through in vitro and in vivo experiments and clinical data analysis, we verified that Src was involved in neutrophil‐mediated innate immune responses, further confirming the regulatory effects of Src on inflammatory immunity. The gene discussed is SRC; the disease is osteoarthritis.