PRRT2 and COVID-19: Previous studies had shown that PKC inhibitors reduced the proinflammatory and tissue damage effects of neutrophils in COVID‐19.[30] The deficiency of Geranylgeranyl diphosphate synthase in neutrophils could promote the NETs formation through PKC/NOX signaling, thereby exacerbating lipopolysaccharide (LPS) induced lung injury.[31] In Figure S11 (Supporting Information), we found through the classical NETs model induced by PMA in vitro that inhibiting PKC significantly reduced the NETs formation.