The analysis of negatively correlated pathways indicated that endocytosis (Endocytosis, ES = −0.3992, NP = 0.002) and natural killer cell-mediated cytotoxicity (ES = −0.5098, NP = 0.012) were statistically significant (Fig 8B), suggesting that BAAT might participate in the IPF process by regulating immune responses. The gene discussed is BAAT; the disease is idiopathic pulmonary fibrosis.