CCR2 and congenital bilateral aplasia of vas deferens from CFTR mutation: The chemokine receptor CCR2 mediates monocyte egress from the bone marrow and monocyte recruitment to sites of inflammation and strongly contributes to atherogenesis.5 In individuals with cardiovascular risk, monocyte CCR2 expression is positively correlated with arterial wall inflammation.6 Studies on the involvement of CCR2 in CAVD are scarce.