Mollaoglu's study supports this notion, proposing that in the transformation from adenocarcinoma to squamous carcinoma, the deletion of NKX2‐1 or overexpression of SOX2 recruits tumour‐associated neutrophils, with the CXCL5 chemokine further promoting SOX2‐driven squamous cell transformation.58 This evidence concerns the gene NKX2-1 and neoplasm.