Several potential mechanisms may explain this association: (i) m6A-mediated regulation of the critical key enzymes of the Warburg effect or aerobic glycolysis: Wang et al.‘s study demonstrated that METTL3 enhances the stability of one of the critical enzymes for the Warburg effect, HK2, through YTHDF1-mediated m6A modification, thereby promoting the Warburg effect in cervical cancer [32]. The gene discussed is HK2; the disease is cervical cancer.