Furthermore, BACH1 activates the transcription of HK2 (Hexokinase 2) and GAPDH (glyceraldehyde-3-phosphate dehydrogenase), enhancing glucose uptake, glycolytic flux, and lactate secretion, thereby promoting lung cancer metastasis through a glycolysis-dependent pathway [169–171]. This evidence concerns the gene GAPDH and lung carcinoma.