These results are consistently explained by pituitary damage after tumor resection and/or sequelae of hypothalamic compression by the tumor, without contradiction—even for the paradoxically mildly elevated PRL, as well as the persistently and markedly low baseline levels of GH, TSH, LH, and FSH, and the mildly reduced ACTH observed after two pituitary tumor resections. This evidence concerns the gene GH1 and neoplasm.