NFKB1 and Alzheimer disease: PB has been shown in animal models of Alzheimer's disease to reduce neuroinflammation and oxidative stress by increasing GSH levels, inhibiting lipid peroxidation and ROS production, and lowering the levels of pro‐inflammatory cytokines (TNF‐α, IL‐6, IL‐1β, and IL‐1α), MDA, iNOS, NF‐κB, COX‐2, and NO.