While clinically 50% of patients with T-LGL leukemia have neutropenia, with 20% as severe neutropenia and 15%–39% as infections, they also display peripheral lymphocytosis, which our patient did not, and their bone marrow is typically hypercellular, not hypocellular like in this case, with usually less than 50% infiltration by CD3+, CD8+, and CD57+ clonal LGL cells (6). Here, CD8A is linked to infection.