There were three different groups of mice utilized for this study; In the control group, mice were administered with Tamoxifen to affect the epithelial deletion of DCLK1 (these are the uninfected controls), CR (mice were administered with tamoxifen and given oral CR infection to induce colitis), CR+DBZ (mice were administered tamoxifen, given oral CR infection and DBZ; Dibenzazepine, a γ-secretase inhibitor). This evidence concerns the gene DCLK1 and colitis.