ACSL1 and neoplasm: Consistently, both the oncogenes (EGFR, BRCA1, CDC20, etc.)[73–75] and the pro-oncogenic genes (PIK3R6, PXN, FZD2, etc.)[76–78] were inhibited, while the genes (ACSL1, GADD45G, CRB3, etc.)that inhibit tumor progression [79–81] were up-regulated (Fig. 6M).