We observed that, in samples from patients with adenomyosis, the infiltration rates of CD56 dim natural killer cells, effector memory CD8 T cells, eosinophils, immature B cells, myeloid-derived suppressor cells (MDSCs), neutrophils, and plasmacytoid dendritic cells were significantly higher, suggesting that these immune cells play a key role in the progression of adenomyosis (Figure 8A). This evidence concerns the gene NCAM1 and adenomyosis.