Other mechanisms of PRDX5 regulation include reversible thiol modifications by S‐sulphenylation, S‐sulphinylation, S‐glutathionylation, S‐nitrosylation, phosphorylation, and acetylation.[16, 35] There is a report that WSB1 appears to be an oncogene that increases cancer development and thus results in poor prognosis.[21] This discrepancy could potentially be explained by differences in experimental conditions, such as different cell models used. The gene discussed is WSB1; the disease is cancer.