The importance of EndMT in the pathogenesis of DCM is observed in diabetic human hearts, and this process illustrated upregulation of lncRNAs ZFAS1 and MALAT1 while decreasing several miRs (miR‐9, miR‐146a, and miR‐200b) in diabetic patients compared with non‐diabetic patients [169]. The gene discussed is MALAT1; the disease is familial dilated cardiomyopathy.