Additionally, miR-182 inhibitors can reduce cell proliferation promoted by METTL3 overexpression to a certain extent, and METTL3 is required for DGCR8-regulated pri-miRNA in PCa [88], as the reader protein complex recognizes and processes pri-miRNA, thereby producing mature miRNA that affects cellular processes [89]. Here, METTL3 is linked to posterior cortical atrophy.