Bai et al. (2021) conducted research on the functions of the lncRNA MIAT in the mouse model of MI; their findings reveal that MIAT silencing induces reductions of the translocation protein (TSPO) at both the mRNA and protein levels in MI. Indeed, such silencing prevents mitochondrial permeability transition pore opening, thereby safeguarding the integrity of the mitochondrial membrane from hypoxic damage (Bai et al., 2021). This evidence concerns the gene MIAT and myocardial infarction.