CCL28 and colorectal carcinoma: (2023) showed that bile salt hydrolase (BSH) activity in non-enterotoxigenic Bacteroides potentiated colorectal cancer (CRC) by increasing colonic deoxycholic acid (DCA) and lithocholic acid (LCA), which activated tumor cell TGR5, upregulated CCL28, and promoted immunosuppressive Treg infiltration.