However, increased free N‐termini were detected in resis, NAA15‐RNAi, and NAA10‐RNAi plants, which is consistent with previous observations of loss‐of‐function mutants of the NatA complex.[31, 32] Additionally, after pathogen infection, the number of free N‐termini did not decrease in the three transgenic plants, indicating that the function of the NatA complex during the pathogen response was suppressed under RESIS knockout conditions, similar to NAA15 or NAA10 knockout (Figure 5a). Here, NAA15 is linked to infection.