Pathogens are known to affect host protein synthesis, and the host mounts a defense response by controlling the translation process, leading to an increase in the transcription of immune genes.[66] Our results revealed that translation increased after pathogen infection in WT plants and that translation was further increased by RESIS or NAA15 knockdown, suggesting that the RESIS‐NatA complex regulates resistance through the modulation of translation (Figure 7g). This evidence concerns the gene NAA15 and infection.