The PB2 WT cells formed larger and more heterotypic clusters with WBCs than PB2 KO cells without affecting tumor cell viability (Fig. 5b–c, and Supplementary Fig. S4b, S8a), demonstrating that PLXNB2 promotes heterotypic tumor-WBC cluster formation in which tumor cells can possibly evade immune cell killing. The gene discussed is PLXNB2; the disease is neoplasm.