Concurrently, apoptotic caspases can cleave cGAS and MAVS, thereby inhibiting the activation of both cGAS and RIG‐I‐like receptors initiated innate immune responses.[70, 72] Given the inhibitory effect of coronavirus on cell death activation, we hypothesized that this inhibition might promote IAV replication and exacerbate infection‐induced inflammatory responses. Here, CGAS is linked to infection.