These findings are in line with the reported anti‐inflammatory effects of the STING inhibitor H‐151 in experimental dermatitis [62], and with the protective role of STING deficiency against hyperglycemia‐induced NF‐κB activation and oxidative mitochondrial damage in endothelial cells in vivo [63]. Here, STING1 is linked to hyperinsulinemic hypoglycemia, familial, 4.