ETV1 and posterior cortical atrophy: In fact, while the established PNT2‐derived cells with de novo ETV1 or ERG overexpression constitute clear cell models to identify ETS‐driven oncogenic pathways, with ETS overexpression being introduced as the single driver of the acquisition of a malignant phenotype [10, 43, 44], the cell models LNCaP and VCaP, with underlying ETV1 and ERG rearrangements/overexpression, respectively, constitute ETS‐driven models of advanced PCa, providing insightful validation of the therapeutic potential of targeted approaches.