In this regard, Beedanagari et al. [84], based on the results obtained in MCF7 breast cancer cells in which resveratrol significantly reduced dioxin-induced CYP1A1 expression, suggested that the resveratrol mechanism of action involves inhibition of the recruitment of transcription factors AhR and ARNT recruitment to the xenobiotic response elements (XREs) of the corresponding genes. This evidence concerns the gene AHR and breast carcinoma.