Although sepsis also induces hepatic expression and systemic accumulation of another 12 kDa protein, serum amyloid A (SAA) [25,26], our anti-human-pro-DCD pAbs did not react a peptide (e.g., P9) that shares some homology with human SAA (Figure 1E), further supporting the possibility of pro-DCD induction in rodents during severe infections. The gene discussed is DCD; the disease is Sepsis.