To further explore this relationship and evaluate the impact of SC, alone or in combination with antileukemic agents, on chemoresistant cells, we examined the expression levels of equilibrative nucleoside transporter 1 (ENT1) and deoxycytidine kinase (dCK), two genes known to sensitize AML cells to Ara-C [3,30,31]. The gene discussed is DCK; the disease is acute myeloid leukemia.