TNF and Alzheimer disease: Other pathways regulated by the differently expressed miRNAs were apoptosis, synaptic function of glutamatergic, cholinergic, and GABA-ergic synapses and long-term potentiation, as well as TNF- and IL-17 signaling, or cytokine–cytokine receptor interaction [149], highlighting the involvement of neuroinflammation in the pathogenesis of AD [88].