Additionally, some other scores, such as genetic diffusion and transcriptomic diffusion, are also high in both forms (Figure 4b), which shows that SOD1 interacts with several targets that either have genetic links to the disease or exhibit differential expression in ALS (Figure 4c), allowing comprehensive assessment of SOD1′s role in ALS pathology in patients with specific genetic mutations. This evidence concerns the gene SOD1 and amyotrophic lateral sclerosis.