Indeed, hs-cTnT was able to rate chronic myocardial injury 5 times more than hs-cTnI (20% vs. 4%) while still having similar performance in identifying acute myocardial injury (hs-cTnT 14% and hs-cTnI 15%), with a greater prognostic value for all-cause mortality, incident MI, revascularization, or hospitalization due to heart failure in the convalescent period after an episode of acute chest pain (hs-cTnT/I HR 1.38 vs. 1.14) [23] in a population of 48–73 year old patients. This evidence concerns the gene TNNT2 and heart failure.