In this study, we showed that overexpression of DNAJC7 promotes the disassembly of arsenite-induced TDP-43 condensates in a U2OS cell model (Fig. 5d, e), suggesting that overexpressed DNAJC7 may attenuate ALS pathophysiology by controlling the misfolded TDP-43. The gene discussed is DNAJC7; the disease is amyotrophic lateral sclerosis.