In addition, CXCL9 and CXCL10, which share the common receptor CXCR3 (mainly expressed on the surface of immune cells, such as T cells and NK cells [162]) have been reported to adversely contribute to MASLD; serum CXCL9 was shown to be upregulated in patients with biopsy-proven MASH compared to controls, and hepatic CXCL9 was shown to be upregulated in MCD-diet fed mice compared to WT mice [163]. This evidence concerns the gene CXCR3 and metabolic dysfunction-associated steatotic liver disease.