Similarly, CXCL10 was shown to be higher in the liver and serum of patients with histologically confirmed NASH compared to patients with simple hepatic steatosis and controls, as well as in the liver of MCD-diet fed mice, in which CXCL10 promoted hepatic lipogenesis, inflammation, and oxidative stress [164]. This evidence concerns the gene CXCL10 and Hepatic steatosis.