The activation of tumor‐intrinsic STING signaling leads to IFN production, which facilitates the recruitment of DCs, T cells, and macrophages to the tumor microenvironment, thereby promoting MHC expression and enhancing tumor antigen presentation.[6] Our findings indicated that P2 promoted intratumoral infiltration of CD4+ and CD8+ T cells, and enhanced the recruitment of DCs with potent antigen‐presenting capacity within the tumor microenvironment. The gene discussed is IFNA1; the disease is neoplasm.