For example, LINC00114 and CCN2 have previously been found to be upregulated in B‐ALL compared to T‐ALL while deletions of EBF1 have been associated with B‐ALL [112, 113] and VPREB3 has been found as a methylation and expression signature gene between B‐ and T‐ALL [111]. Here, EBF1 is linked to acute lymphoblastic leukemia.