In addition, a proteomic study of non‐ NSCLC cells‐derived EVs identified HSPA1A, MFGE8, and ANXA4 as mediators of tumor‐recipient cells crosstalk.[40] While these molecules could play roles in other contexts, our experimental evidence strongly supports the dominance of miR‐485‐3p in this specific model. This evidence concerns the gene ANXA4 and non-small cell lung carcinoma.