Moreover, in another study showed that GA inhibited the expression of SREBP-1c and LXR α, key transcription factors that regulate fatty acid and triglyceride synthesis, while also downregulating the fatty acid transporter CD36, gene expression of fatty acid transport protein (FATP 2) and ACCα attenuated FFA uptake and lipogenesis, thereby improving hepatic steatosis. This evidence concerns the gene SREBF1 and fatty liver disease.