METTL3 exerts a positive regulatory effect on pri-miR-BART3-3 p through its interaction with the microprocessor protein DGCR8 in an m6A-dependent manner, leading to a reduction in PLCG2 that promotes the proliferative capacity and tumor growth of NK/T cell lymphoma (NKTCL) cells in vitro and in vivo [86]. Here, METTL3 is linked to extranodal nasal NK/T cell lymphoma.