ENO1 and colorectal carcinoma: Results obtained from EdU cell proliferation assays, colony formation assays, CCK8 assays, PDO models, and the CRC subcutaneous xenograft mouse model showed that ENO1 overexpression reversed the inhibition of CRC cell growth induced by LINC00183 silencing, while ENO1 knockdown significantly attenuated the pro-proliferative phenotype driven by LINC00183 overexpression (Fig. 8A–D, E and G)(Supplementary Fig. S7A–D).