JUN and melanoma: TF showed anti‐proliferative and pro‐apoptotic effects on A375 human melanoma cells in concentration‐dependent manner via P53 and JNK pathwaysTF markedly stimulated proteins associated with the P53 pathway (ATM, CHK1/2, P53, and CASP8/3) and those linked to the JNK pathway (ASK1, JNK, and C‐JUN) via phosphorylation and cleavage, leading to the activation of pro‐apoptotic factors (PARP, BAX, BIM, PUMA, and P53)