SELE and atherosclerosis: This process is tightly regulated by adhesion molecules (e.g., ICAM, VCAM, E-selectin and P-selectin) that undergo upregulated expression during atherosclerosis (Richardson et al., 1994; Poston et al., 1992; Johnson-Tidey et al., 1994; Nakashima et al., 1998; Li et al., 1993).