In general, HeV-g1-infected AGMs exhibited greater peak log2FC values for analytes associated with chemotaxis (Eotaxin, IL-8, IP-10, MCP-1, and MIP-1beta) whereas HeV-g2 infection produced increased levels of fibrinolysis and endothelial damage markers (tissue factor, factor IX, P-selectin, tPA, sCD40L, PSGL-1, PAI-1). The gene discussed is CCL4L2; the disease is infection.