Additionally, LNT enhances the antitumor effects of DLL1 through two immune mechanisms: it promotes the infiltration and activation of CD8+ T cells, thereby activating adaptive immunity, and induces neutrophils to polarize toward the N1 type, secreting cytokines such as IFN-γ to synergize with tumor suppression and remodel innate immunity. The gene discussed is IFNG; the disease is neoplasm.