In this case, urinalysis on admission was pivotal: despite only mild proteinuria (UPCR 0.24 g/gCr) and a lack of findings suggestive of active glomerulonephritis, markedly elevated levels of multiple urinary tubular injury markers (NAG, β2MG, and L-FABP) provided strong evidence for DI-AIN. This evidence concerns the gene FABP1 and glomerulonephritis.