The majority of CRC cases are due to altered KRAS and B‐Raf, which activate WNT‐APC‐CTNNB1, TGFB1‐SMAD, and RAS–RAF–MAPK pathways and promote proliferation with suppressed apoptosis (Ahronian et al. 2015). Here, CTNNB1 is linked to colorectal carcinoma.