NFKB1 and influenza: Notably, we observed an induced enrichment of specific lung bacteria, particularly a marked increase in the genus Staphylococcus, which has been previously identified as a potential contributor to secondary pneumonia in altered lung microenvironments following influenza infection.[63] Furthermore, the activation of the NF‐κB pathway, a primary inflammatory response route, has been associated with pneumonia and damage caused by Methicillin‐resistant Staphylococcus aureus.