Specifically, we provide an in‐depth analysis of BBR's mechanisms of action—including its impact on neuroinflammation, oxidative stress, mitochondrial dysfunction, and specific molecular pathways (e.g., PI3K/Akt, AMPK, NF‐κB, lncRNAs)—as they relate to various neurodegenerative conditions such as Alzheimer's, Parkinson's, multiple sclerosis (MS), stroke, spinal cord injury (SCI), Huntington's, and other forms of dementia. This evidence concerns the gene NFKB1 and Stroke.