Specifically, we provide an in‐depth analysis of BBR's mechanisms of action—including its impact on neuroinflammation, oxidative stress, mitochondrial dysfunction, and specific molecular pathways (e.g., PI3K/Akt, AMPK, NF‐κB, lncRNAs)—as they relate to various neurodegenerative conditions such as Alzheimer's, Parkinson's, multiple sclerosis (MS), stroke, spinal cord injury (SCI), Huntington's, and other forms of dementia. The gene discussed is NFKB1; the disease is stroke disorder.