RBM11 and neoplasm: This modulation may promote tissue repair and regeneration (e.g., activation of the Wnt/β-catenin pathway to enhance epithelial or bone regeneration; miR-126-mediated vascular protection; and induction of M2 macrophage polarization to suppress inflammation), or, conversely, facilitate pathological signaling (e.g., RBM11-mediated alternative splicing promoting tumor proliferation and drug resistance, or hepatocyte-derived ApoBDs activating fibrogenic pathways in hepatic stellate cells).