HDAC4 and Duchenne muscular dystrophy: HDAC4‐dependent molecular mechanisms of neuronal death could be different, for example: (1) excitotoxic glutamate, by increasing HDAC4, activates caspase 3 and induces neuronal apoptosis [22], whereas (2) in a murine model of Duchenne muscular dystrophy, HDAC4 increasing and activation of receptor‐interacting serine/threonine‐protein kinase 1 (RIPK1) determines necroptotic cell death [23].