IFNG and hepatocellular carcinoma: This study further demonstrates that intratumoral MAIT cells from HCC patients upregulate the expression of PD-1+, CTLA-4+, and TIM-3+ inhibitor or exhaustion markers along with diminished cytotoxic molecules, including IFN-γ, granzyme B, and perforin, in comparison to MAIT cells from peritumor regions.