On the other hand, the Gαt [G38D] mutant responsible for the Nougaret form of night blindness showed a near-normal GTP-binding, and a slight reduction in both intrinsic and RGS9-stimulated GTPase activity (Muradov and Artemyev, 2000), although this study employed a Gαt/Gαi1 chimera comprised of 94% of Gαt residues. Here, GLYAT is linked to night blindness.