The dual PI3K/mTOR inhibitor NVP‐BEZ235 has demonstrated potential in overcoming hypoxia‐ and TGF‐β1‐induced EMT in OC and PCa by downregulating HIF‐1α and its related molecules, including Snail and phosphorylated Smad2/3, resulting in the reinstatement of E‐cadherin and a decrease in cellular motility. Here, PIK3CB is linked to posterior cortical atrophy.